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When activated, it drives production of TNF-alpha, IL-6, IL-1 beta, and dozens of other inflammatory mediators

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N-acetylcysteine is a precursor to glutathione

WB and chromatin immunoprecipitation (ChIP) analyses revealed that p53 overexpression can target and negatively regulate biological activity by binding to the SLC7 A11 promoter [35], indicating that p53 can mediate ferroptosis through multiple pathways at the same time