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MELAS is not the result of a single gene defect and as such represents a constellation of related disorders that can result from defects in several of the mitochondrial genome encoded proteins of complex I

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Mechanistically, H2S regulates intracellular O2 homeostasis and inhibits hypoxia-induced HIF-1 activation in a VHL- and mitochondria-dependent manner (Kai et al., 2012)