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These enhancements were supported by DFT calculations, which confirmed that lattice strain and sulfur vacancies increased Na affinity and accelerated electron transfer

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In NAD + -depleted states (e.g., aging or advanced disease), SIRT1 activation consumes residual NAD + , impairing NAD + -dependent repair enzymes such as PARPs, and increasing oxidative stress

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In addition, BPC-157 can improve muscle strength and endurance