Unger, "GCG is the key factor in the development of diabetes," Diabetologia, vol
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doi: 10.2174/0929867327999200716112353 64
In this study, we find that the GSH-deficient mutant, in spite of the highly accumulated Glu previously defined as a critical wound signal essential for activating plant defense events, showed impaired plant defense signaling events (Fig
Since the sequence homology between exenatide and human GLP-1 is only 53%, 40% to 60% of patients develop antiexenatide antibodies, though in most cases antibody development does not reduce the efficacy of exenatide treatment [44,49-51]