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Recent studies suggest that some NSAIDs directly inhibit DPP4, thus elevating endogenous GLP-1 [19]

Goal: Stimulate endogenous growth hormone release to support anabolic processes related to muscle growth, fat metabolism, and tissue repair [1] [2]

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[10] noticed doubling the risk of cholecystectomy among patients treated with GLP-1RAs

38 Studies 39 have shown that GERD patients with lower HRV values exhibit a decrease in parasympathetic components and an increase in sympathetic components, indicating vagal dysfunction

doi: 10.1039/D2FO01096H