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In particular, a reduced folate intake would be an important determinant of carotid stenosis related to hyperhomocysteinemia (Selhub et al, 1995), and a decrease in plasma folate concentration below 13.6 nmol/L would be associated with a gradual increase in the risk of coronary artery disease (Morrison et al, 1996

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Tendon healing In a rat model in the Journal of Applied Physiology , BPC-157 accelerated tendon healing by promoting tendon-cell outgrowth, cell survival and cell migration, which the authors linked to the VEGF and nitric-oxide pathways [2]