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Mutation frequencies were 2- to 3-fold higher in the Ogg1 -/- mice than in wild type Of the 16 base substitutions detected in Ogg1 -/- mutant plaques analyzed by sequencing, 10 indicated G:C-A:T transversions consistent with the known spectrum of mutation The results support that insufficient repair of oxidized bases leads to mutation

These findings highlight the potential of GHRP-6 as a therapeutic candidate for mitigating the cardiotoxic and systemic side effects of anthracycline chemotherapy while supporting overall survival [1]

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As we age, glutathione levels in the body gradually decrease, which can lead to increased oxidative stress and premature aging

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