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IGF-1 LR3 provides direct, rapid, IGFBP-bypass IGF-1R activation appropriate for acute molecular signalling research, GH-independent muscle biology, and insulin-resistance-dissociated anabolic research
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The No DAC version does not contain the Drug Affinity Complex (DAC) modification, resulting in a shorter-acting research analogue
Cancer 85 , 428430 (2001)
Protective effects of the mechanistic target of rapamycin against excess iron and ferroptosis in cardiomyocytes