This dual antioxidant mechanism explains GHK-Cu's protective effects in inflammatory conditions and its ability to enhance cellular survival under stress conditions
maintain reconstituted peptide at 28 C and use within 7 days
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The peptide counteracted picrotoxin (non-competitive GABA receptor Cl channel blocker), isoniazid (inhibitor of glutamic acid decarboxylase and GABA synthesis), and bicuculline (GABA-A antagonist), all through dose-dependent protective effects without producing behavioral changes when administered alone