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This paradox underscores the complexity of FASNs role in EC biology, suggesting that both excessive and insufficient DNL can contribute to endothelial dysfunction, depending on the context and temporal dynamics

Diabetes Care 47 (11), 18731888

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Proteins downregulated by GLP-1GIPLani (cluster 1) were mainly associated with calcium and tubulin binding, ion channel and phosphatase regulation and protein folding, whereas proteins upregulated by GLP-1GIPLani (cluster 3) were linked to oxidative and lipid metabolism, ribosomal activity and amino acid and carbohydrate metabolism (Fig