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& Utermann, G

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By potentially restraining Caspase-3 activity, Epithalon might support cellular longevity and decrease apoptosis, enhancing its regenerative capabilities

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Alternative and non-mutually exclusive mechanisms may involve favorable effects on basal energy expenditure [57] or on the choice of less energy-rich foods consumed in free-living conditions [58], although these latter hypotheses remain to be validated in clinical studies

Se deficiency has been associated with autoimmune thyroid diseases (AITD) and sepsis, arteriosclerosis, cardiovascular disease, cancer, increased mortality among the elderly and hemodialysis patients, and cognitive decline [5, 6]