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This insulin-independent mechanism provides glucose lowering that is less dependent on beta-cell function, though the glycaemic effect diminishes with declining renal function

The latter is consistent with our own findings of increased GSH concentrations in treated hypertensive patients (9)

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Impairment of the intestinal barrier facilitates the translocation of bacteria and microbial metabolites, such as IS and LPS, into systemic circulation, inducing endotoxemia ( Figure 2 ) [112]

Human tumor-associated mutations in BAP1 do not inhibit the expression of SLC7A11 and do not suppress tumors via promoting ferroptosis [50]

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