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Description

STING/ACSL4 axis-dependent ferroptosis and inflammation promote hypertension-associated chronic kidney disease

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Nevertheless, advances in neuroimaging, biomarker analysis, and longitudinal cohort studies have enabled researchers to corroborate key mechanistic pathways in humans, particularly those involving neurotrophic deficits, hippocampal atrophy, and neurotransmitter dysregulation

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doi: 10.1107/S090744490901169X Escoubas, Pierre and King, Glenn F

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TM, thrombomodulin

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