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Subsequent sections examine the core mechanismsanti-apoptotic, angiogenic, antioxidative, immunoregulatory and epigeneticthrough which MSCs reprogramme the damaged ovary, and we evaluate complementary strategies such as cell-free exosomes, small-molecule pre-conditioning and bio-engineered artificial ovaries

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Hansotia T, Baggio LL, Delmeire D et al (2004) Double incretin receptor knockout (DIRKO) mice reveal an essential role for the enteroinsular axis in transducing the glucoregulatory actions of DPP-IV inhibitors