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GPX4-dependent chondrocyte ferroptosis in OA GPX4 has recently attracted more attention in OA due to its ability to eliminate lipid peroxides and inhibit ferroptosis [23]

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These observations indicate that in presence of TLR2 targeting peptide, TLR2 pep-orid-liposome is capable more extendedly of accumulating and retaining in AML, thereby efficiently delivering drug to the target site