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Scheckhuber et al., 2007)

Imaging studies show structural and functional CNS alterations, highlighting CNS dysfunction as a key pain driver

Silencing of Wls in hUCMSCs suooressed -catenin activation and HFSCs proliferation

To address this, we employed a sequential, multi-omics bioinformatics workflow to generate a testable mechanistic hypothesis, followed by rigorous experimental validation

This avoids the subsequent supersensitivity of the dopamine receptors and the raised amphetamine disturbances