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Next, we measured the cristae width of hundreds of mitochondria and determined that rapamycin-treated CAR T cell products had significantly looser mitochondrial cristae, which is consistent with their less-activated metabolic state (Fig

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Cysteine is not a substrate but a specific modulator of human ASCT2 (SLC1A5) transporter

An integrated and unifying hypothesis for the metabolic basis of sporadic Alzheimers disease