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In lung cancer cells, activation of GPR81 decreases intracellular cyclic adenosine monophosphate (cAMP) levels and inhibits protein kinase A (PKA) activity, leading to activation of Transcriptional co-activator with PDZ-binding motif (TAZ), which further activates the programmed cell death protein 1/programmed death-ligand 1 (PD-L1/PD-1) immune checkpoint pathway and impairs T-cell function

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