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To address the question of the relative contribution to antioxidant electron flux we made use of genetic or chemical inhibition of the glutathione and thioredoxin-dependent pathways and monitored the cytosolic NADP redox dynamics in response to exogenous oxidants in living cells and tissues

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Common impurities include truncated sequences (particularly des-Gly1 and des-Val15 variants), deamidation products at Asp residues, and residual TFA from synthesis

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