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Understanding which signaling pathways produce GH release versus cortisol release versus appetite stimulation could enable the rational design of compounds that activate only the desired downstream effects

This is also consistent with the hypothesis that MGF is acting on neural progenitor cell proliferation followed by differentiation and maturation initiated by IGF-1

Ipamorelin selectively triggers the pituitary to release GH more frequently without elevating cortisol or prolactin

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And the best part

Ipamorelin is designed to primarily stimulate growth hormone release without significantly impacting other hormones like cortisol or prolactin