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RelA/p65 promotes osteoclast differentiation by blocking a RANKL-induced apoptotic JNK pathway in mice

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1a, Supplementary Fig

That adipose-specific NNMT knockdown protects against diet-induced obesity in mice through mechanisms involving increased adipocyte energy expenditure

It is a synthetic version of a hormone called glucagon-like peptide-1 (GLP-1), which is produced naturally in the body

Scientific References GLP-1 receptor agonists and dual GLP-1/GIP receptor agonists: strengthened warnings on acute pancreatitis including necrotising and fatal cases