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Inhibition of GCL with buthionine sulfoximine (BSO) depleted glutathione rapidly, reflecting its short half-life (1-4 h) as a substrate of multiple enzymes (Fig

We hypothesized that the effective intracellular GSH levels of the gsh-px deficient mutant were higher than that of the parental strain, resulting in enhanced virulence of the deletion strain

These slightly red shifts were due to the addition of CeO x to hinder the oscillation of phosphorus atoms of BP, leading to the red shift of these typical Raman peaks and reducing the corresponding Raman scattering energy of BP@CeO x nanozymes (Supplementary Fig

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