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In pancreatic cells, activation of these G proteincoupled receptors is primarily mediated through pathways involving cyclic adenosine monophosphate (cAMP), protein kinase A ( PKA ), and cAMP response elementbinding protein ( CREB ), thereby enhancing glucose-dependent insulin secretion and supporting -cell function

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Combined or Multi-Target Approaches Due to the complex feedback within the iron-senescence-fibrosis triad, combination therapies targeting multiple pathways may be more effective than single-agent treatments

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