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Homozygous and mutated SLC19A3 gene can cause various conditions such as biotin-thiamine responsive basal ganglia disease (BTBGD), lactic acidosis combined with encephalopathy, infantile epileptic spasms, and early childhood encephalopathy induced by disease or trauma

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It has been observed that patients affected by systemic lupus erythematosus are colonized by a more homogeneous intestinal microbiota [1, 74, 75, 95]

The lack of these receptors ensures GI macrophages are unable to respond to ligands that would typically activate other immune cell types thereby preventing excessive IRs (Brenchley and Douek 2012)

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