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From a mechanistic perspective, the contraction of muscle fibers and the hemodynamic forces elicited by exercise activate a complex network of intracellular signaling pathways which collectively enhance mitochondrial biogenesis and quality control, stimulate angiogenic processes, and augment eNOS activity along with NO production (73, 74)

It is associated with increased menstrual irregularity, ovulatory dysfunction, development of insulin resistance and infertility

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doi:10.1111/j.1471-4159.2010.06731.x 115