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The peptide acts as an agonist at the ghrelin/GHS receptor, stimulates Gq and Go signaling, increases intracellular calcium in somatotrophs, triggers exocytosis of GH-containing vesicles, and may also stimulate GHRH release and reduce somatostatin at the hypothalamic level

[4] In other words, most patients with detectable anti-sermorelin antibodies continue to respond to therapy, and the antibodies may disappear over time even if treatment continues

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Interestingly, some of the natural sulfur compound such as cysteine, GSH, and taurine are also intermediates of TSP, which is a fundamental metabolic pathway that maintains the redox buffer in cells

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