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& Zakhary, M

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It fosters heterogeneity in outcome measures, mechanisms of action, and temporal scales, blurring distinctions between true neuroprotection, symptomatic relief, and disease modification

Under glucose deprivation or increased ATP demand, AMPK activation is expected to shift oligodendrocytes from an anabolic, myelin-maintenance program toward a catabolic state that supports survival

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[DOI] [PubMed] [Google Scholar] 71.Li J, Kreicbergs A, Bergstrom J, Stark A, Ahmed M