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The glucose-dependent mechanism means that when blood sugar levels are normal, insulin secretion is not excessively stimulated, providing a safer metabolic profile than some older diabetes treatments
This leads us to pose the question of how we develop species-specific understanding that clearly defines human pathology, enabling the targeted development of treatments in humans
Human prostate cancer (PCa) cell lines LNCaP and C4-2 were maintained in RPMI 1640 medium supplemented with 10% FBS
Beyond direct antioxidant work, glutathione plays a core part in toxin clearance
Nat Med 8(7):738742 Ggebakan , Andres J, Biedasek K et al (2012) Glucose-dependent insulinotropic polypeptide reduces fat-specific expression and activity of 11-hydroxysteroid dehydrogenase type 1 and inhibits release of free fatty acids