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Consistently, exogenous H 2 S was unable to prevent apoptotic injury in cells expressing Syvn1 C115A under HG + PA condition (Fig

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Recent finding in TBK1 deficiency mice suggested that loss of TBK1 in microglia causes an aging-like inflammatory state in these cells

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This not only serves as additional energetic resource, but also generates a number of biologically active molecules such as diacylglycerol, cholesterol, ceramide, sphingosine, PIP2, IP3 which are involved in the activation of a variety of signaling pathways associated with cancer progression, metastases and therapy resistance e.g

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