Obesity is a chronic relapsing disease process, attributable to the complex interaction between genes and environmental risk factors, characterised by excess adiposity, that adversely impairs health [13]
Its ability to stimulate collagen synthesis, glycosaminoglycan production, and balanced tissue remodeling is supported by multiple studies
Furthermore, another study [38] reported that tuna backbone protein hydrolysate showed strong hydroxyl radical scavenging activity (80.91% at a concentration of 1.3 mg/ml)
Biological Functions of Selenium in Neurological Disease It is estimated that 25 selenoproteins are present in the human body at present, including glutathione peroxidase1 (GPX1), glutathione peroxidase2 (GPX2), glutathione peroxidase3 (GPX3), glutathione peroxidase4 (GPX4), glutathione peroxidase6 (GPX6), thioredoxin reductase1 (TrxR1), thioredoxin reductase2 (TrxR2), thioredoxin reductase3 (TrxR3), iodothyronine deiodinases1 (DIO1), iodothyronine deiodinases2 (DIO2), iodothyronine deiodinases3 (DIO3), methionineRsulfoxide reductase B1 (SelR), selenophosphate synthetase 2 (SPS2), selenoproteins F (SelF), selenoproteins H (SelH), selenoproteins I (SelI), selenoproteins K (SelK), selenoproteins N (SelN), selenoproteins M (SelM), selenoproteins O (SelO), selenoprotein P (SelP), selenoproteins V (SelV), selenoproteins S (SelS), selenoproteins T (SelT), and selenoproteins W (SelW) [16, 17]
In human clinical trials, a marked increase in glutathione levels was shown regardless of its initial (basal) concentration
B.ZhaiP.DobleA.TaylorC
