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Beilmann, A.Portmann, University of Zrich, Institute of Pharmacology and Toxicology) highlights how sections versus conventional secondary antibody-based IHC

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Collectively, these observations militate against the idea that AA-induced alpha cell proliferation accounts for the high circulating levels of glucagon observed acutely upon administration of GRAs, an effect which we instead attribute to the removal of GLP-1(936)s suppressor effect