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Abstract Background: The widespread clinical use of immune checkpoint inhibitors (ICIs) in oncology has been accompanied by an increased incidence of immune-related adverse events (irAEs)
2024;10:e28457
2 expression is highly upregulated by exogenous GSH supplementation indicating a substrate-dependent regulatory mechanism (Ghosh et al., 2022)
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YA were released after 2-weeks, but OA continued supplementation (c)
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