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RE progression results in damage to intestinal epithelial cells, the perturbation of tight junction proteins and an imbalance of microbes, leading to conditions favorable for the enrichment of Enterococcus 16

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This evidence has inspired the ligandin model for vacuolar sequestration of anthocyanins ( 1 ), in which arGSTs are believed to protect the labile anthocyanidins ( 3 ) from degradation and/or guide them from their biosynthetic origin on the cytoplasmic surface of the endoplasmic reticulum to transporters located on the tonoplast membrane 18,19,31

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All research cited refers to preclinical and in vitro studies

Ober, R