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The IARC TP53 Database: New Online Mutation Analysis and Recommendations to Users
[78] found that cells were highly sensitive to peroxide-induced ferroptosis by replacing selenocysteine in GPX4 with cysteine, demonstrating the importance of selenium for GPX4 to function as an antioxidant against ferroptosis
Miller WH, Jr
Advanced degenerative conditions severely degenerated cartilage or end-stage joint disease involves tissue that may be beyond repair acceleration
A disturbance in this balance can result in excessive ECM degradation or accumulation, leading to lung dysfunction and disease progression
Kalirin-7 controls activity-dependent structural and functional plasticity of dendritic spines
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