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Three mechanistic explanations, not mutually exclusive: Signalling cascade persistence: BPC-157 activates VEGFR2 and FAK-paxillin pathways whose downstream effects (angiogenesis, fibroblast migration, collagen remodelling) unfold over days to weeks after initial receptor engagement

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The pulsatile nature of GH release induced by this combination, rather than continuous elevation, has been identified as a key factor in maintaining receptor sensitivity and avoiding desensitization artifacts that can complicate long-term research protocols