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Phosphorylates Bad and other pro-apoptotic proteins preventing mitochondrial outer membrane permeabilization (point of no return in intrinsic apoptosis)
Hence, this autoantibody presence especially targets the autonomic and central nervous systems (503), which may explain the dysautonomia and immune dysregulation present in ME/CFS (58)
Given the potentially broad reparative effects, IL-22 agonism represents a highly rational target in AH
Secondly, GLP-1 can also potentiate the release of insulin relative to glucose absorption, hence improving glucose clearance from the blood
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