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Studies have shown that JAK2 may also phosphorylate insulin receptor substrate 1/2 (IRS1/2) and activate the phosphatidylinositol 3-kinase/protein kinase B (PI3K/AKT) pathway (Ridderstrale et al., 1995

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This mechanism provides a valuable research model for investigating aging biology, cell-cycle regulation, and the role of senescent cells in tissue function