Mechanism ColonBroom's GLP-1 positioning is backed by real fiber science: fermentable fiber increases short-chain fatty acids, which activate receptors on gut L-cells that release GLP-1 and PYY, both of which increase fullness
L-carnitine and other trimethylamine (TMA) containing compounds (including GBB and crotonobetaine) can be further metabolized by gut bacteria to yield free TMA (Koeth et al., 2014
Furthermore, their synthesis is limited to some species, they were exclusively found in mammals (also basic ones such as platypus) except cattle [124] and dogs [125]
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Nam HJ, Jang IH, You H, Lee KA, Lee WJ (2012) Genetic evidence of a redox-dependent systemic wound response via Hayan protease-phenoloxidase system in Drosophila
The following articles explain your genetic susceptibility variants related to phase II detoxification, nutrients and cofactors needed for optimal support, and how to prioritize what is most important for you, personally, to avoid.[ref] Nrf2 Signaling Pathway master switch inducing the phase II enzyme pathways, upregulated by sulforaphane [ref] UGT genes glucuronidation, most prevalent phase II enzyme GST genes glutathione conjugation reactions SULT genes sulfation reaction increases the water solubility of drugs, hormones, and xenobiotics
