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Meanwhile, NRF2 activated heme oxygenase-1 (HO-1) expression, which encouraged Fe 2+ buildup in LIP and iron release from hemoglobin, as well as iron apoptosis development

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We found that APR-017 or LCS3 treatment induced an increased level of ROS and a decreased level of GSH (presented as the ratio of GSH to oxidized form, GSSG) in VRK2-KO cells, but not VRK2-WT cells (Fig

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(191): In CKD rats, ferroptosis inducer cisplatin (CP) reduced GSH and GPX4, while inhibitor desferrioxamine (DFO) elevated these markers, confirming ferroptosis modulation as an RF intervention