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Its design enables selective receptor binding to GLP-1, GIP, and glucagon receptors to optimize metabolic impact balanced with side effects

Using the acetate form would mean having to constantly reconstitute fresh batches to ensure consistent potency, introducing more steps and more room for error

Seizure-induced reduction in glucose utilization promotes brain hypometabolism during epileptogenesis

[29], who found significantly enhanced postprandial GLP-1 release at 60 and 120 min after meals at both 6 and 12 months post-LSG

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