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A novel Osmium-based compound targets the mitochondria and triggers ROS-dependent apoptosis in colon carcinoma

The slow titration schedule exists specifically to minimize these effects

Whether the deglutathionylation requires a release or reorientation of the GSSR intermediate should depend on whether the four sulfur atoms of the disulfide substrate, GSH, and the active-site cysteinyl residue can be arranged like beads on string

Thus, NSPs released from activated neutrophils may promote both primary tumors and metastatic tumor expansion

Evidence note: The candid clinician's position is to hold both truths at once

The supplemental data contain a list of these important works