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Endothelial dysfunction and prothrombotic reprogramming Endothelial cells (ECs) exposed to NET components undergo profound functional reprogramming, shifting from physiological vascular homeostasis toward inflammationdriven endothelial injury and a procoagulant phenotype [78]

Preclinical studies on related polyphenols demonstrate their potential: quercetin nanoparticles showed enhanced antioxidant activity (88), liposomal silymarin improved hepatoprotective efficacy, and phytosomal curcumin exhibited greater absorption (89)

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While these mechanisms are well-documented in general skin-brightening studies, the application for those with localized depigmentation is more complex