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Examining the expression levels of ferroptosis-related genes in angiographically determined coronary artery disease patients

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In hepatocellular carcinoma, glutathione S-transferase Pi (GSTP) translocates to the nucleus and modulates redox signaling via the Nrf2-Keap1 complex

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The data robustly suggest that despite its role in reducing the ERs GSHpool, CHAC1 does not influence the UPR, implying that GSHdepletion alone may not be sufficient to disrupt protein folding homeostasis to a degree that activates the UPR