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ALOX12 serves as the primary gene responsible for driving ferroptosis, and its mechanism of action involves the conversion of polyunsaturated fatty acids, particularly arachidonic acid, into the bioactive lipid (12S)-hydroperoxide eicosapentaenoic acid (Zheng et al., 2020)

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While collagen is a component of cartilage extracellular matrix, GHK-Cus effects appear more pronounced in skin and connective tissue rather than cartilage specifically

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CYP2E1 activation from heavy drinking diverts hepatic resources away from cannabinoid metabolism, while acetaldehyde accumulation adds toxic burden