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Mitochondrial injury in AP is accompanied by decreased m, elevated ROS levels, activation of the NLRP3 inflammasome, and pyroptosis, ultimately leading to PAC necrosis ( This interaction is likely to be especially important in HTG-AP, in which overwhelming delivery of triglyceride-derived non-esterified fatty acids (NEFAs) creates a lipotoxic environment that exceeds the oxidative capacity of a single organelle system

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Most pronounced during the first few weeks and after each dose escalation