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failure[43] due to oxidative stress regularly appears as a result of the lysis of endothelial cells[9,10], and it may be further aggravated by additional reperfusion

Studies have shown superiority of this version over the others

To address these limitations, recent studies have successfully achieved in vivo mtDNA editing by delivering DdCBEs to the mouse heart using adeno-associated viral (AAV) vectors

The lowest tier at 1 mg per week served primarily to assess tolerability, producing modest weight reduction outcomes