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doi: 10.3389/fnut.2020.598286 68 MorataA.LoiraI.EscottC.del FresnoJ

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Unlike acute wounds, which follow a well-defined healing cascade of re-epithelialization, fibroblast proliferation, and angiogenesis, chronic wounds remain trapped in a prolonged inflammatory phase characterized by microbial persistence and dysregulated immune responses [4,5,6]

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This is why it is often discussed alongside broader categories such as GLP-1 agonists, metabolic peptides, and non-insulin diabetes research