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Based on the negative regulatory effects of HDAC11 on PAI-1, the use of HDAC11 agonists is very likely to protect I/R-induced renal injury by upregulating the expression of HDAC11 and inhibiting the expression of PAI-1, thus becoming a potential therapeutic strategy for I/R-induced AKI

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These observations strongly implicate microglia in disease progression, though whether their activation primarily drives pathology or represents a protective response remains debated

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