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However, knockdown or deletion of TET2 shifts the gene expression profiles of these myeloid cells toward a pro-inflammatory phenotype, impairing their suppressive capabilities and promoting anti-tumor immune responses [100]

10.2967/jnumed.121.262300 191 HorsagerJ.KnudsenK.SommerauerM

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Dying to defend: neutrophil death pathways and their implications in immunity

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