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The standard treatment for vitamin B12 deficiency involves administering cobalamin (either hydroxo- or cyanocobalamin) via intramuscular injection

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In contrast, ferroptosis inhibition via iron chelation or lipid radical scavenges blocked this Hmox1 activation-induced cardiac damage, supporting a ferroptosis targeting therapeutic strategy to alleviate cardiomyopathy in SCD 46

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It is also important to test our approach on P